Skip to content

Large plaque parapsoriasis: clinical and genotypic correlations

J Cutan Pathol. 2000 Feb;27(2):57-60. Large plaque parapsoriasis: clinical and genotypic correlations. Simon M(1), Flaig MJ, Kind P, Sander CA, Kaudewitz P. Large plaque parapsoriasis: Clinical and genotypic correlations on ResearchGate, the professional network for scientists. Large plaque parapsoriasis: clinical and genotypic correlations. Clinically, among the patients with clonal disease one developed clearcut mycosis fungoides (MF) after a follow-up of 8 years, in the other 5 patients no such diagnosis could be made after follow-up of 2-21 years (median: 9 years).

Large plaque parapsoriasis: clinical and genotypic correlations 2A patient with clinicopathologic features of small plaque parapsoriasis presenting later with plaque-stage mycosis fungoides: report of a case and comparative retrospective study of 27 cases of nonprogressive small plaque parapsoriasis. Large-plaque parapsoriasis (LPP) and small-plaque parapsoriasis (SPP) are recognized. Simon M et al: Large plaque parapsoriasis: Clinical and genotypic correlations. Professional medical resources for Parapsoriasis including Large plaque parapsoriasis: clinical and genotypic correlations.

Comparison with large plaque parapsoriasis and benign chronic dermatoses. Kaudewitz, P. Large plaque parapsoriasis: clinical and genotypic correlations. Parapsoriasis is considered to be a cutaneous lymphoprolifer. Kind P, et al. Large plaque parapsoriasis: clinical and genotypic correlations. Kaudewitz P. Large plaque parapsoriasis: clinical and genotypic correlations.

Similar Articles For Pubmed (select 10438729)

Retrospective study of 24 patients with large or small plaque parapsoriasis treated with ultraviolet B therapy 310 Simon M, Flaig MJ, Kind P, Sander CA, Kaudewitz P. Large plaque parapsoriasis: clinical and genotypic correlations. J Cutan Pathol 2000; 27: 57-60. Psoriatic plaque thickness is a clinical measure of psoriasis severity. Thick plaques associated with male gender, increased body mass index, nail disease, psoriatic arthritis, larger plaques, more body sites, and greater total body surface area affected. The Utah Psoriasis Initiative (UPI) was established to study and classify subtypes of psoriasis based upon observable and patient-reported characteristics to facilitate the correlation of psoriasis phenotypes with genotype. Several genetic correlations between psoriasis and IBD have been reported thanks to Genome Wide Association Studies (GWAS) that have identified 13 psoriasis susceptibility loci (called PSORS1-13) and 28 IBD susceptibility loci (called IBD1-28) (Figure 1). Bernardini et al., Plaque Psoriasis: Anatomical, Clinical and Immunohistochemical Correlations During Anti-Tnfa Treatment, vol., CARD15/NOD2 mutational analysis and genotype-phenotype correlation in 612 patients with inflammatory bowel disease, The American Journal of Human Genetics, vol. They are classified according to clinical morphology, site and extent of involvement as well as the predominating epidermal structure within the individual lesion. Skin lesions are linear, pruritic, reddened and hyperkeratotic papules or plaques. May resemble psoriasis. Large epidermal naevi, especially in the head and neck, may merit CNS assessment. Should patients with psoriasis receive vitamin D supplementation?

References In Defining Early Mycosis Fungoides