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CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris

CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris 1

Psoriasis vulgaris is a common skin disorder characterised by focal formation of inflamed, raised plaques that constantly shed scales derived from excessive growth of skin epithelial cells. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Potential role of the chemokine receptors CXCR3, CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Lab Invest 2001 Mar;81(3):335-47J B Rottman, T L Smith, K G Ganley, T Kikuchi, J G Krueger. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris.

CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris 2Title: Potential role of the chemokine receptors CXCR3, CCR4, and the integrin alpha E beta 7 in the pathogenesis of psoriasis vulgaris. Source: LABORATORY INVESTIGATION 81 (3): 335-347. Potential role of the chemokine receptors CXCR3, CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Lab Invest. 2001 Mar; 81(3):335-47. Psoriasis vulgaris is a common skin disorder characterised by focal formation of inflamed, raised plaques that constantly shed scales derived from excessive growth of skin epithelial cells. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris.

Buslau, M., Jochum, W. et al, T cells involved in psoriasis vulgaris belong to the Th1 subset. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Potential role of the chemokine receptors CXCR3, CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. CrossRef; PubMed. Lab. Potential role of the chemokine receptors CXCR3, CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Laboratory investigation; a journal of technical methods and pathology 2001; 81: 335 47.

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CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Since colonization and infection by bacteria can exacerbate psoriasis,3,4 we sought to determine the cellular immunological reactivity pattern for prepsoriatic (PN) skin (also known as symptomless skin) in the absence and presence of various bacteria-derived products, such as superantigens, lipopolysaccharide (LPS), and heat shock proteins (HSPs), using a skin-explant assay system. T lymphocytes derived from skin lesions of patients with psoriasis vulgaris express a novel cytokine pattern that is distinct from that of T helper type 1 and T helper type 2 cells. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Psoriasis vulgaris is one of the most prevalent chronic inflammatory skin diseases affecting approximately 2 of individuals in Western societies, and found worldwide in all populations. CCR4, and the integrin alphaEbeta7 in the pathogenesis of psoriasis vulgaris. Psoriasis vulgaris is one of the most prevalent chronic inflammatory skin diseases affecting approximately 2 of individuals in Western societies, and found worldwide in all populations.

References In From Laboratory To Clinic: Rationale For Biologic Therapy